[1] Arweiler, N.B. and L. Netuschil, The oral microbiota. Microbiota of the human body: implications in health and disease, 2016: p. 45-60.
[2] Mutha, N.V., et al., Transcriptional profiling of coaggregation interactions between Streptococcus gordonii and Veillonella parvula by Dual RNA-Seq. Scientific reports, 2019. 9(1): p. 7664.
[3] Sedghi, L., et al., The oral microbiome: Role of key organisms and complex networks in oral health and disease. Periodontology 2000, 2021. 87(1): p. 107-131.
[4] Mohammed, W.K., N. Krasnogor, and N.S. Jakubovics, Streptococcus gordonii Challisin protease is required for sensing cell--cell contact with Actinomyces oris. FEMS microbiology ecology, 2018. 94(5): p. fiy043.
[5] Jakubovics, N.S., Intermicrobial interactions as a driver for community composition and stratification of oral biofilms. Journal of molecular biology, 2015. 427(23): p. 3662-3675.
[6] Davey, M.E. and G.A. O'toole, Microbial biofilms: from ecology to molecular genetics. Microbiology and molecular biology reviews, 2000. 64(4): p. 847-867.
[7] Bowen, W.H., et al., Oral biofilms: pathogens, matrix, and polymicrobial interactions in microenvironments. Trends in microbiology, 2018. 26(3): p. 229-242.
[8] Kolenbrander, P.E., et al., Communication among oral bacteria. Microbiology and molecular biology reviews, 2002. 66(3): p. 486-505.
[9] Jakubovics, N.S., S.A. Yassin, and A.H. Rickard, Community interactions of oral streptococci. Advances in applied microbiology, 2014. 87: p. 43-110.
[10] Choo, S.W., et al., Transcriptomic Responses to Coaggregation between Streptococcus gordonii and Streptococcus oralis. Applied and Environmental Microbiology, 2021. 87(22): p. e01558-21.
[11] Contreras, A., et al., Periodontal microbiology in L atin A merica. Periodontology 2000, 2015. 67(1): p. 58-86.
[12] Parahitiyawa, N., et al., Exploring the oral bacterial flora: current status and future directions. Oral diseases, 2010. 16(2): p. 136-145.
[13] Do, T., et al., Population structure of Streptococcus oralis. Microbiology, 2009. 155(Pt 8): p. 2593.
[14] Hirschfeld, J. and T. Kawai, Oral inflammation and bacteremia: implications for chronic and acute systemic diseases involving major organs. Cardiovascular & Haematological Disorders-Drug Targets (Formerly Current Drug Targets-Cardiovascular & Hematological Disorders), 2015. 15(1): p. 70-84.
[15] Chalmers, N.I., et al., Characterization of a Streptococcus sp.-Veillonella sp. community micromanipulated from dental plaque. Journal of bacteriology, 2008. 190(24): p. 8145-8154.
[16] Cross, B.W. and S. Ruhl, Glycan recognition at the saliva–oral microbiome interface. Cellular immunology, 2018. 333: p. 19-33.
[17] Yoshida, Y., et al. Streptococcal receptor polysaccharides: recognition molecules for oral biofilm formation. in BMC Oral Health. 2006. Springer.
[18] Yadav, R.K. and V. Krishnan, New structural insights into the PI‐2 pilus from Streptococcus oralis, an early dental plaque colonizer. The FEBS Journal, 2022. 289(20): p. 6342-6366.
[19] Xu, H., et al., Streptococcal co-infection augments Candida pathogenicity by amplifying the mucosal inflammatory response. Cellular Microbiology, 2014. 16(2): p. 214-31.
[20] Diaz, P.I., et al., Using high throughput sequencing to explore the biodiversity in oral bacterial communities. Molecular Oral Microbiology, 2012. 27(3): p. 182-201.
[21] Palmer, R.J., Jr., et al., Mutualism versus independence: strategies of mixed-species oral biofilms in vitro using saliva as the sole nutrient source. Infection Immunology, 2001. 69(9): p. 5794-804.
[22] Luo, A., et al., Formation, development, and cross-species interactions in biofilms. Frontiers in Microbiology, 2022. 12: p. 3982.
[23] Lapa, S., et al., Multiplex PCR for identification of bacterial pathogens of infectious pneumonia. Russian journal of bioorganic chemistry, 2020. 46: p. 859-861.
)
