NKP30-B7-H6 Interaction Aggravates Hepatocyte Damage through Up-Regulation of Interleukin-32 Expression in Hepatitis B Virus-Related Acute-On-Chronic Liver Failure
Fig 3
Up-regulated hepatic IL-32 expression in HBV-ACLF patients.
IL-32 expression was detected in liver samples from patients with HBV-ACLF (Fig 3, A–G, original magniifcation 200x), mild CHB (Fig 3H, original magnification 200x), and healthy controls (Fig 3I, original magnification 200x) by immunohistochemical staining. Relative mean density analysis indicated that the hepatic B7-H6 expression in HBV-ACLF patients was significantly higher than that in mild CHB patients or healthy controls (P < 0.05) (Fig 3J).