Cellular targets for the beneficial actions of tea polyphenols1234
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From Charité–Universitätsmedizin Berlin, Medizinische Klinik für Kardiologie und Angiologie, CCM, Berlin, Germany.
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Presented at the conference “Fifth International Scientific Symposium on Tea and Human Health,” held at the US Department of Agriculture, Washington, DC, 19 September 2012. The conference was organized by Jeffrey Blumberg, Tufts University, Boston, MA, and a Steering Committee including representatives from each of the symposium cosponsors: the American Cancer Society, the American College of Nutrition, the American Institute for Cancer Research, the American Medical Women’s Association, the American Society for Nutrition, and the Linus Pauling Institute. The symposium was underwritten by the Tea Council of the USA. Its contents are solely the responsibility of the authors and do not necessarily represent the official views of the Tea Council of the USA or the cosponsoring organizations.
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ML received an honorarium and travel support from the Tea Council of the USA for speaking at the Fifth International Scientific Symposium on Tea and Human Health and for preparing this article for publication.
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Address correspondence to M Lorenz, Charité–Universitätsmedizin Berlin, Medizinische Klinik für Kardiologie und Angiologie, Campus Mitte, Kardiologisches Forschungslabor, Hessische Straße 3-4, 10115 Berlin, Germany. E-mail: [email protected].
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Abbreviations used: EGCG, epigallocatechin-3-gallate; eNOS, endothelial nitric oxide synthase; ERα, estrogen receptor α; FITC, fluorescein isothiocyanate; miRNA, microRNA; mRNA, messenger RNA; NO, nitric oxide; ROS, reactive oxygen species; TF3, theaflavin-3,3′-digallate; 67LR, 67-kDa laminin receptor.