An Intronic SINE Insertion in FAM161A that Causes Exon-Skipping Is Associated with Progressive Retinal Atrophy in Tibetan Spaniels and Tibetan Terriers
Figure 5
Graphical comparison of the intron-exon boundaries of FAM161A.
A) Human (Homo sapiens) FAM161A. B) Canine FAM161A exons confirmed by sequencing the retinal mRNA transcript. The location of the splice site insertion is indicated.