DPP4 Deficiency Exerts Protective Effect against H2O2 Induced Oxidative Stress in Isolated Cardiomyocytes
Figure 5
Effects of H2O2 treatment on ERK and AKT phosphorylation in wild-type and DPP4-deficient cardiomyocytes, with or without GLP-1 or GLP-1 receptor antagonist cotreatment.
(A) Original western blots of ERK and p-ERK were reported. (C) Ratios of p-ERK/ERK were shown. (B) Original western blots of AKT and p-AKT were reported. (D) Ratios of p-AKT/AKT were shown. (n = 3–4) *p<0.05 vs. wild-type control, #p<0.05 vs. DPP4-deficient control, ‡p<0.05 vs. wild-type H2O2, &p<0.05 vs. wild-type H2O2 + GLP-1.