Skip to main content
Advertisement
Browse Subject Areas
?

Click through the PLOS taxonomy to find articles in your field.

For more information about PLOS Subject Areas, click here.

< Back to Article

Renalase Deficiency in Heart Failure Model of Rats—A Potential Mechanism Underlying Circulating Norepinephrine Accumulation

Figure 1

Protein expression of renalase diminished in the ischemic compared to the non-ischemic kidney.

A, There is no statistic difference between renalase expression between the ACEI and control groups both in the ischemia kidney and the nonischemia one. B, Western blotting showing renalase protein expression of ischemia kidney is lower than that of the nonischemia one. Alpha-tubulin also shown as a housekeeping control. Corresponding graph shows accumulated results from n = 8 (A) or n = 16 (B) for each group (0.295±0.085 versus 0.765±0.171). *p<0.01 vs. ischemic kidney. NS: No statistical difference is existed between the two groups.

Figure 1

doi: https://doi.org/10.1371/journal.pone.0014633.g001