Developmental Cell
Volume 39, Issue 1, 10 October 2016, Pages 1-2
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A TRIM16-Galectin3 Complex Mediates Autophagy of Damaged Endomembranes

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Selective autophagy, an essential process for maintaining intracellular homeostasis, depends on precise target recognition and local activation. Reporting in Developmental Cell, Chauhan et al. (2016) elegantly demonstrate that interaction between TRIM16 and Galectin3 orchestrates the recruitment of core autophagic factors and activates selective autophagy at the site of damaged endomembranes.

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