Mechanisms of endotoxin shock and endotoxin hypersensitivity
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Lipopolysaccharide Structure and the Phenomenon of Low Endotoxin Recovery
2022, European Journal of Pharmaceutics and BiopharmaceuticsCitation Excerpt :Typically, when large quantities of these bioactive mediators are circulated in the body, it can result in high fever, hypotension and septic shock.[14] However, it is important to recognise that in some instances when the host organism is hyper-reactive to LPS, the production of even low concentrations of mediators can be fatal.[2,4]. With LPS being known as the primary factor responsible for the toxicity associated with Gram-negative bacteria,[15] much research is being invested into how to eliminate and neutralise LPS, which may be present via contamination, as well as harnessing the potential of LPS to act as a potent immunomodulator.[12]
Improved bioluminescence-based endotoxin measurement method using a salt-resistant luciferase mutant
2021, Analytical BiochemistryEicosanoids derived from cytochrome P450 pathway of arachidonic acid and inflammatory shock
2019, Prostaglandins and Other Lipid MediatorsHepatoprotective effects of AdipoRon against D-galactosamine-induced liver injury in mice
2016, European Journal of Pharmaceutical Sciences
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Dr. Chris Galanos, Max-Planck-Institut für Immunbiologie, Stübeweg 51, D-7800 Freiburg, Germany
Copyright © 1993 Gustav Fischer Verlag · Stuttgart · New York. Published by Elsevier GmbH All rights reserved.