Muscle damage induced by experimental hypoglycemia
References (25)
Hemiplegia in a diabetic producing unilateral peripheral neuritis and hypoglycemic attacks
Lancet
(1967)- et al.
Hypoglycemia and angina
Lancet
(1985) - et al.
How many creatine kinase “isoenzymes” are there and what is their clinical significance?
Clin Chim Acta
(1981) Separation of tissue and serum creatine kinase isoenzymes on polyacrylamide gel slabs
Clin Chim Acta
(1972)- et al.
Liver enzyme abnormalities after insulin induced hypoglycemic coma
BMJ
(1988) - et al.
A rise in the plasma activities of hepatic enzymes is not a common consequence of hypoglycemia
Diabet Med
(1988) - et al.
Liver function tests after hypoglycemia in insulin-dependent diabetic patients
Practical Diabetes
(1988) - et al.
Plasma hepatic glutathione S-transferase concentrations after insulin-induced hypoglycemia in normal subjects and diabetic patients
Diabet Med
(1989) - et al.
Plasma enzymic changes in insulin-induced hypoglycemia in experimental rabbits
Tohoku J Exp Med
(1996) Studies on CK isoenzyme analysis
Jpn J Clin Pathol
(1976)
Creatine kinase
Jpn J Med Technol
Hypoglycemia in the Diabetes Control and Complications Trial
Diabetes
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Atrazine is primarily responsible for the toxicity of long-term exposure to a combination of atrazine and inorganic arsenic in the nigrostriatal system of the albino rat
2013, Neurotoxicology and TeratologyCitation Excerpt :In order to evaluate the possible systemic damage to liver and muscle, the serum of nine animals per group was used to determine aspartate transaminase (AST) (WeinerLab, Rosario, Ar), creatine kinase (CK), and lactate dehydrogenase (LDH) (Stanbio, Boerne, TX, USA). The combined use of these markers provides general evidence of liver and skeletal muscle damage in humans (Jiang et al., 1998) and in animal models (Nathwani et al., 2005). The appropriate kits were used following the manufacturer's specifications.
Glutathione suppresses increase of serum creatine kinase in experimental hypoglycemia
2007, Diabetes Research and Clinical PracticeCitation Excerpt :Whether acute hypoglycemia in diabetic patients might cause immediate and significant raises in serum ALT, AST and alkaline phosphatase (ALP) activities, as a sign of hepatic damage has been controversial [4,17–19]. In our previous studies [11–13], we demonstrated a remarkable rise in plasma ALT, AST, LDH, and CK activities by hypoglycemia, which are primarily due to damage in skeletal and/or heart muscle rather than liver, and that change in plasma GSSG induced by hypoglycemia might be related to increase of serum enzymes. Further, we studied on the alteration of plasma levels of glutathione in order to explicate mechanism of the rise in serum enzymes caused by experimental hypoglycemia, and demonstrated significant increase of oxidized glutathione.
Surveilling brain damage using brain biomarkers in hypoglycemic neonatal calves with diarrhea
2023, Frontiers in Veterinary ScienceClinical analysis of the serum muscle enzyme spectrum of patients with newly diagnosed Sheehan's syndrome
2022, Medicine (United States)