Cranial nerve deficit: A clue to the diagnosis of ethylene glycol poisoning
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Cited by (19)
Ethylene glycol poisoning: Resolution of cranial nerve deficit
2014, Journal for Nurse PractitionersCitation Excerpt :MRI revealed enhancement of cranial nerves IX, X, and XII and enhancement of the right cochlear nerve. These radiologic findings may be seen in the setting of EG toxicity.2-5 The patient was started on a course of prednisone 60 mg by mouth, daily for 7 days, followed by a 10 mg/d taper for 7 days (totaling 14 days).
Methanol and Ethylene Glycol Intoxication
2012, Critical Care ClinicsCitation Excerpt :In fatal cases, death most often develops during this phase. If the patient survives the neurologic and cardiopulmonary manifestations, severe acute renal failure often develops, marking the third stage of ethylene glycol poisoning, which typically develops 24 to 72 hours after ingestion.67,69,82,90,99,122,123,130,139,141–143 The acute renal failure can be prolonged enough to necessitate hemodialysis, sometimes continuing long after the patient otherwise recovers.99,130
Early and Late Presentations of Ethylene Glycol Poisoning
2009, American Journal of Kidney DiseasesCitation Excerpt :Incorporating serum ethanol measurement (if available) into the calculated serum osmolality would reflect the contribution of ethylene glycol to the increased osmolal gap. Cranial neuropathies previously have been reported as delayed sequelae of ethylene glycol poisoning, with onset from 8 to 18 days after significant ingestion (up to 900 mL), especially in patients who presented late to hospitals.29-36 Patient 2 developed a right seventh cranial nerve palsy about 14 days after ingestion of ethylene glycol.
Characteristics, Pathophysiology, and Effects of Common Toxic Substances
2008, Critical Care Nephrology, Second EditionToxicology in the critically ill patient
2003, Clinics in Chest MedicineAdult toxicology in critical care part II: Specific poisonings
2003, ChestCitation Excerpt :Stage 3 (2 to 3 days) is dominated by acute renal failure due to acute tubular necrosis with an element of tubular obstruction from calcium oxalate precipitation.484950 Late (6 to 18 days) neurologic sequelae have also been described in survivors.5152 Methanol is metabolized by alcohol dehydrogenase to formaldehyde, which is converted by aldehyde dehydrogenase to formic acid.