International Journal of Radiation Oncology*Biology*Physics
Volume 29, Issue 1, 30 April 1994, Pages 133-139
Hyperthermia original contributionHyperthermia radiosensitization in human glioma cells comparison of recovery of polymerase activity, survival, and potentially lethal damage repair
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Cited by (28)
Effects of hyperthermia as a mitigation strategy in DNA damage-based cancer therapies
2016, Seminars in Cancer BiologyCitation Excerpt :It has been postulated that the latter hypothesis is more likely to occur due to their higher detection levels [11]. Evidence from several hyperthermia-induced radio-sensitization studies has shown that heat induction can affect DNA repair pathways as it restricts replication by reducing the activity of DNA polymerases, thus eventually increasing the appearance of more DNA breaks after irradiation [12–14]. In addition, data from early research reports suggested that heat treatment can promote topoisomerase inactivation in HeLa S3 cells [15] while other studies conducted in human epidermoid cancer (KB) cells have reported increased transcription and translation levels of topoisomerase II after exposure to 42 °C or 45 °C.
Thermal radiosensitization by protracted low temperature hyperthermia in human glioma cells
1996, Journal of Thermal BiologyThe DNA Double-Strand Break Repair in Glioma: Molecular Players and Therapeutic Strategies
2022, Molecular NeurobiologyHealing of the cancer tissues under the action of moving heat and non-local Caputo–Fabrizio heat transport
2022, Waves in Random and Complex MediaRole of Laser Interstitial Thermal Therapy in the Management of Primary and Metastatic Brain Tumors
2021, Current Treatment Options in Oncology
Copyright © 1994 Published by Elsevier Inc.