Review paperComplement activation and cytokine production as consequences of immunological bioincompatibility of extracorporeal circuits
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Blood compatibility of nanomaterials
2018, Drug Delivery Nanosystems for Biomedical ApplicationsProtein adsorption to polyethylene glycol modified liposomes from fibrinogen solution and from plasma
2001, Biochimica et Biophysica Acta - BiomembranesCitation Excerpt :The other complement proteins investigated, i.e., factors B, H, and I were all depleted relative to plasma. This finding argues against significant complement activation since factor B is known to bind to C3 when activation occurs [41] so that factor B would be expected to be enriched on the liposome surface. The cell-adhesive proteins fibronectin and vitronectin were also investigated.
Ellipsometric in vitro studies on the activation of complement by human immunoglobulins M and G after adsorption to methylated silicon
2001, Colloids and Surfaces B: BiointerfacesClinical performance of a new high-flux synthetic membrane
2000, American Journal of Kidney DiseasesCitation Excerpt :Our study has shown minimal activation with both the materials studied, and the total absence of activation may be a consequence of surface interactions of other still unidentified factors with glycoproteins and phospholipids contained in the cell membrane. Complement activation induces interleukin-1 and tumor necrosis factor-α production in monocytes, which is enhanced in the presence of the small amounts of lipopolysaccharides known to be present in dialysis fluid.34,35 C5b-9 attacks such autologous nonnucleated cells as red blood cells and may contribute to the structural and functional erythrocyte changes seen in renal failure and the shortened erythrocyte survival times.36,37