Research reportGABAA receptor stimulation enhances NMDA-induced Ca2+ influx in mouse cerebral cortical neurons in primary culture
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2016, Pharmacology Biochemistry and BehaviorCitation Excerpt :GABAA receptor regulates glutamate release from a population of neocortical glutamatergic neurons via their own presynaptic receptors (Long et al., 2009). For example, GABAA receptor stimulation has been shown to contribute to protective mechanisms involved in neuronal injury induced by NMDA receptor activity (Ohkuma et al., 1994). Numerous evidence supports the notion that GABAA receptor function in the brain is structured via several calcium-regulated mechanisms which are varied in terms of duration, influx source, and intracellular pathway (Gallos et al., 2015; Sametsky et al., 2015).
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