Editorial
The heart in anaphylaxis

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References (33)

  • NAM Paterson et al.

    Release of chemical mediators from partially purified human lung mast cells

    J Immunol

    (1976)
  • A Pomerance

    Periarterial mast cells in coronary atheroma and thrombosis

    J Path Bacteriol

    (1958)
  • R Levi et al.

    Effects of histamine on the cardiovascular and respiratory system: dysrrhythmias caused by histamine release in guinea pig and human hearts

    Klin Wochenschr

    (1982)
  • R Levi et al.

    Leukotriene C4 is released from the anaphylactic heart: a case for its direct negative isotropic effect

  • R Levi et al.

    Acetylglyceryl ether phosphorylcholine (AGEPC): a putative mediator of cardiac anaphylaxis in the guinea pig

    Circ Res

    (1984)
  • N Capurro et al.

    The heart as a target organ in systemic allergic reactions: comparison of cardiac anaphylaxis in vivo and in vitro

    Circ Res

    (1975)
  • Cited by (46)

    • Atrial Fibrillation in Anaphylaxis

      2017, American Journal of Medicine
      Citation Excerpt :

      In these cases, the very interesting point of discussion was: which should be first, the treatment of the anaphylaxis or of the cardiac event? Treatment with H1 antagonists is controversial; in fact, these drugs prevent slow atrioventricular conduction and inhibit ventricular fibrillation,4 but they are considered safe. Corticosteroids are potent anti-inflammatory and immunosuppressant agents.

    • The Use of Vasopressors and Inotropes in the Emergency Medical Treatment of Shock

      2008, Emergency Medicine Clinics of North America
      Citation Excerpt :

      Shock occurs in 30% to 50% percent of cases [132,133]. Shock in anaphylaxis shares variable components with hypovolemic shock caused by capillary fluid leak, distributive shock caused by the loss of vasomotor tone, and cardiogenic shock caused by inotropic reductions [131–134]. Knowledge of this physiologic distribution is important to the emergency management of anaphylaxis and specifically to the selection of therapies.

    • Anaphylactic shock: The advantages of intra aortic balloon counter pulsation for the treatment of heart failure

      2007, Resuscitation
      Citation Excerpt :

      The low arterial pressure and consequent myocardial ischaemia combined with the vasoconstrictive action of the epinephrine could explain the appearance of the abnormal T waves and ST segment.5 Even in subjects without coronary disease the administration of adrenaline results in abnormal signs in the ST segment, suggestive of heart failure.6 In anaphylactic shock the systematic vasoplegia may induce severe arterial hypotension.

    • Anaphylaxis: A review of causes and mechanisms

      2002, Journal of Allergy and Clinical Immunology
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