[38] Recycling of insulin-sensitive glucose transporter in rat adipocytes
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Insulin signaling meets vesicle traffic of GLUT4 at a plasma-membrane-activated fusion step
2005, Cell MetabolismCitation Excerpt :The cytosol was analyzed to assess the amount of depletion prior to use in the fusion assay. Basal and insulin-stimulated plasma membranes were prepared (Simpson et al., 1983) and reconstituted in phospholipid liposomes using an adaptation of previously described methods (Kono, 1983; Schurmann et al., 1989). Purified plasma membrane was solubilized in 1.25% n-octyl glucoside and applied to Sephadex G50 column.
MKK6/3 and p38 MAPK Pathway Activation Is Not Necessary for Insulin-induced Glucose Uptake but Regulates Glucose Transporter Expression
2001, Journal of Biological ChemistryCitation Excerpt :Glucose transport is known to be affected by cell differentiation and oncogenic transformation, as well as by various hormones and growth factors. Such modulation has often been attributed to altered expression of one or more glucose transporter isoforms (i.e.GLUT1–6) (1, 2) or to modulation of glucose transporters' (particularly GLUT4) capacity to translocate to the plasma membrane (38-40). The current study was undertaken to understand better the role of MKK6/3-p38 MAPK activation on the regulation of glucose transport and glucose transporter expression.
Quantitative methods for measuring the insulin-regulatable glucose transporter (Glut4)
1997, Journal of Pharmacological and Toxicological MethodsAssays for Insulin and Insulin-Like Activity Based on Adipocytes
2015, Drug Discovery and Evaluation: Pharmacological Assay, Fourth Edition