Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis
A reviewThe tumor suppressive properties of adeno-associated viruses
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Analysis of hepatic and retinal cell microRNAome during AAV infection reveals their diverse impact on viral transduction and cellular physiology
2020, GeneCitation Excerpt :The results strongly suggests AAV infection cause host-specific microRNA mediated cell cycle alteration. Multiple reports have demonstrated that AAV suppresses the cellular proliferation in tumorigenic cells and induced tumor models (Bantelschaal, 1990; Schlehofer, 1994). The suppressive properties were attributed to the interference of AAV with cell cycle functions (Winocour et al., 1988).
Adeno-associated virus induces apoptosis during coinfection with adenovirus
2007, VirologyCitation Excerpt :Although AAV requires the presence of a helper virus for a fully permissive infection, AAV inhibits Ad replication and gene expression during coinfection (Casto et al., 1967a; Casto et al., 1967b; Jing et al., 2001; Timpe et al., 2006). AAV also acts as a tumor suppressor, restricts Ad-induced cellular proliferation (Khleif et al., 1991; Ostrove et al., 1981), and sensitizes cells to chemotherapeutic agents (Schlehofer, 1994). The AAV genome contains two open reading frames, rep and cap, which encode the nonstructural replication proteins (Rep) and structural capsid proteins (Cap) respectively.
Viral proteins as emerging cancer therapeutics
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