The effect of a neurotoxin (benzalkonium chloride) on the lower esophagus☆
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Cited by (13)
A novel in vivo model of permanent intestinal aganglionosis
2014, Journal of Surgical ResearchCitation Excerpt :Other models of neurogenic dysfunction have been described throughout the alimentary tract. In 1989, Goto and Grosfeld [23] showed that esophageal denervation using BAC mimicked achalasia. Several studies have also described BAC treatment of the colon and rectum to simulate Hirschsprung disease [7–9,11,14,15,24–27].
Myenteric Denervation of the Gut with Benzalkonium Chloride: A Review of Forty Years of an Experimental Model
2019, Canadian Journal of Gastroenterology and HepatologyResearch in pediatric surgery
2018, Rickham's neonatal surgeryMorphological demonstration of a vagal inhibitory pathway to the lower esophageal sphincter via nitrergic neurons in the rat esophagus
2013, Neurogastroenterology and Motility
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Presented at the Annual Meeting of the Association for Academic Surgery, Salt Lake City, Utah, November 16–19, 1988.
- 2
Associate Professor, Department of Surgery Kyushu University, Fukuoka, Japan.
- 3
Lafayette F. Page Professor and Chairman, Department of Surgery, Indiana University School of Medicine; Surgeon-in-Chief, James Whitcomb Riley Hospital for Children, Indianapolis, IN.
Copyright © 1989 Published by Elsevier Inc.