Symposium on the present status of electrocardiographyThe T wave and ventricular repolarization
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Cited by (35)
Adaptation of ventricular repolarization dispersion during heart rate increase in humans: A roller coaster process
2021, Journal of ElectrocardiologyNormal Ventricular Repolarization and QT Interval: Ionic Background, Modifiers, and Measurements
2017, Cardiac Electrophysiology ClinicsCitation Excerpt :Prolongation of the effective refractory period through K+ channel inhibition together with Na+ channel inactivation remains the more efficient mechanism against reentrant arrhythmias.3,4 It has long been recognized that endocardium has longer action potentials than epicardium.1–3 More recently it has been demonstrated that ventricular myocardium is composed of 3 electrophysiologically and functionally distinct cell types:
Transmural electrophysiological heterogeneity, the T-wave and ventricular arrhythmias
2016, Progress in Biophysics and Molecular BiologyDispersion in ventricular repolarization in the human, canine and porcine heart
2016, Progress in Biophysics and Molecular BiologyTransmural Dispersion of Myofiber Mechanics. Implications for Electrical Heterogeneity In Vivo
2007, Journal of the American College of CardiologyCitation Excerpt :TDRepol may exist in vivo, and the reported lack of it may simply reflect technical difficulties associated with obtaining transmural electrical measurements. For example, Van Dam and Durrer (35,36) noted that the spatial and temporal sequence of repolarization cannot be adequately studied by measuring the steepest portion of the T-wave using either bipolar or unipolar electrodes, because repolarization occurs much more slowly and is of lower voltage than depolarization. To overcome this limitation, Spach and Barr (37) measured potential distributions in closed-chest canine in vivo and found that repolarization was earlier in epicardium than endocardium.