Letter to the EditorNeuroprotective effects of resveratrol: Potential mechanisms
References (20)
- et al.
Inhibitory effect of glutamate release from rat cerebrocortical nerve terminals by resveratrol
Neurochem. Int.
(2009) - et al.
Decreased severity of experimental autoimmune encephalomyelitis during resveratrol administration is associated with increased IL-17+IL-10+ T cells, CD4-IFN-γ+ cells, and decreased macrophage IL-6 expression
Int. Immunopharmacol.
(2009) - et al.
Involvement of oxidative pathways in cytokine-induced secretory phospholipase A2-IIA in astrocytes
Neurochem. Int.
(2009) - et al.
Anti-inflammatory mechanisms of resveratrol in activated HMC-1 cells: pivotal roles of NF-κB and MAPK
Pharmacol. Res.
(2009) - et al.
Dietary supplementation with resveratrol reduces plaque pathology in a transgenic model of Alzheimer's disease
Neurochem. Int.
(2009) - et al.
Resveratrol and its analogs: defense against cancer, coronary disease and neurodegenerative maladies or just a fad?
Mutat. Res. Rev. Mutat. Res.
(2008) - et al.
Dietary supplementation of resveratrol attenuates chronic colonic inflammation in mice
Eur. J. Pharmacol.
(2010) - et al.
Ischemia-reperfusion injury: pathophysiology and clinical implications
Eur. J. Trauma Emerg. Surg.
(2007) - et al.
Induction by IL 1 and interferon-gamma: tissue distribution, biochemistry, and function of a natural adherence molecule (ICAM-1)
J. Immunol.
(1986) - et al.
Mechanism for the protective effect of resveratrol against oxidative stress-induced neuronal death in HT22 cells
Free Radic. Biol. Med.
(2010)
Cited by (13)
The binding of resveratrol to monomer and fibril amyloid beta
2012, Neurochemistry InternationalCitation Excerpt :However, there were no typical association and dissociation reactions between RES and immobilized BSA, indicating the different binding specificities of RES and CR. Combined with the purported antioxidant and anti-inflammatory (Zhu et al., 2008) properties of RES, as well as its possible therapeutic effects on AD (Sahebkar, 2010; Karuppagounder et al., 2009; Huang et al., 2011), and lack of adverse effects (Boocock et al., 2007), it is rational to consider RES as a potential therapeutic compound and diagnostic probe in AD. The fluorescence of the benzothiazole dye thioflavin T (ThT) is a well-known test for amyloid fibril formation (Sulatskaya et al., 2011, 2012).
An organotypic spinal cord slice culture model to quantify neurodegeneration
2012, Journal of Neuroscience MethodsCitation Excerpt :To determine whether the ex vivo co-culture system could accurately depict both neurodegeneration and neuroprotection, a rescue experiment was conducted in which slice cultures that had been stimulated with high densities of microspheres were treated with resveratrol. Resveratrol is an anti-oxidant derived from grape extracts (Pasinetti et al., 2011; Sahebkar, 2010; Sovak, 2001), that has been shown to effectively reduce neuronal death through the prevention of oxidative stress (Fukui et al., 2010; Han et al., 2004; Mokni et al., 2007). Representative images of spinal cord sections cultured with a neurotoxic level of microspheres demonstrated visibly less neurodegeneration when treated with resveratrol (Fig. 6A).
The Role of Resveratrol in Mild Cognitive Impairment and Alzheimer's Disease: A Systematic Review
2022, Journal of Medicinal FoodSome candidate drugs for pharmacotherapy of alzheimer’s disease
2021, Pharmaceuticals