Original ReportFatty Acid Amide Hydrolase and Monoacylglycerol Lipase Inhibitors Produce Anti-Allodynic Effects in Mice Through Distinct Cannabinoid Receptor Mechanisms
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Key words
Endogenous cannabinoid
fatty acid amide hydrolase (FAAH)
monoacylglycerol lipase (MAGL)
neuropathic pain
anandamide
2-ararchidonylglycerol (2-AG)
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Supported by the National Institute on Drug Abuse (grants T32DA007027, P01DA009789, P01DA017259, P50DA005274, and R01DA015197).
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