Original articleParathyroid hormone-stimulated development of osteoclasts in cultures of cells from neonatal murine calvaria
References (26)
Parathyroid gland and bone in vitro VI
Dev. Biol.
(1959)Paracrine interactions in bone-secreted products of osteoblasts permit osteoclasts to respond to parathyroid hormone
J. Biol. Chem.
(1984)- et al.
Effect of Fe2+ and ascorbic acid on acid phosphatase from rat bone
Calcif. Tissue Int.
(1982) The supravital staining of osteoclasts with neutral red: their distribution on the parietal bone of normal growing mice, and a comparison with the mutants gray-lethal and hydrocephalus-3
- et al.
Effect of culture on the hormone responsiveness of bone cells isolated1 by an improved sequential digestion procedure
- et al.
Separation of two bone cell populations from fetal rat calvaria and a study of their responses to parathyroid hormone and calcitonin
J. Endocrinol
(1983) - et al.
In vitro formation of osteoclasts from long-term culture of bone marrow mononuclear phagocytes
J. Exp. Med.
(1982) - et al.
Osteoclast formatio from mononuclear phagocytes: role of bone-forming cells
J. Cell Biol.
(1984) Osteoblasts release osteoclasts from calcitonin-induced quiescence
J. Cell Sci.
(1982)- et al.
Calcitonin alters behavior of isolated osteoclasts
J. Pathol
(1982)
Resorption of bone by isolated rabbit osteoclasts
J. Cell Sci.
(1984)
Isolated bone cells
Effects of parathyroid hormone and calcitonin on osteoclast formation in vitro
Endocrinology
(1980)
Cited by (27)
Activation of dimeric glucocorticoid receptors in osteoclast progenitors potentiates RANKL induced mature osteoclast bone resorbing activity
2016, BoneCitation Excerpt :Mature osteoclasts are not formed in bone marrow, but on surfaces of cortical or trabecular bone. Addition of DEX (10− 7 M) to periosteal/endosteal cells isolated from bone surfaces [36,37] resulted in formation of numerous oversized TRAP+ MuOCL when cells were either stimulated with exogenous RANKL (Fig. 3E, left) or with endogenous RANKL induced by OSM (50 ng/ml) (Fig. 3E, right). These data suggest that when mouse osteoclast progenitor cells from BMM, BMC, or calvarial bones are cultured on plastic and exposed to M-CSF + RANKL + GCs, inhibition of osteoclast formation, escape, and increased formation of large, oversized osteoclasts occurs.
Medicinal Chemistry Opportunities in Bone Metabolism and Osteoporosis
1987, Annual Reports in Medicinal Chemistry
Copyright © 1986 Published by Elsevier Inc.