PaperBinding of single-strand DNA to human platelets
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Neutrophil extracellular traps in thrombi retrieved during interventional treatment of ischemic arterial diseases
2019, Thrombosis ResearchCitation Excerpt :Although FH50 correlated positively with WBC count at fibrinogen levels <4 g/l, the correlation was inverted at higher fibrinogen concentrations (n = 20, R2adj = 0.65, pANOVA = 1.9 × 10−4). Because of the known direct [31–35] and indirect (through von Willebrand factor [36]) NET interactions with platelets and the role of platelets in fibrin formation [37], it was of interest to look at platelet content of the clots. Although in this study we did not observe any associations between measured platelet occupancy and NET components, the SEM analysis of cellular components of thrombi retrieved from AIS, CAD and PAD patients revealed a marked difference in their platelet content (sPlt, Fig. 3).
The role of neutrophils in thrombosis
2018, Thrombosis ResearchCitation Excerpt :Extracellular DNA traps promote platelet activation, platelet aggregation and thrombosis (Fig. 2) [36]. In vitro studies demonstrate that platelets bind to DNA and are activated by histones [37], the two components of NETs respectively [38]. Although the exact mechanism of platelet interaction with NETs remains unknown, interaction via the A1 domain of von Willebrand factor (vWF) is proposed as a plausible mechanism by which vWF can bind to histones [39].
Transport of oligonucleotides across natural and model membranes
1994, BBA - Reviews on BiomembranesLactoferrin binding to human platelets
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