Bone matrix and mineral abnormalities in postmenopausal osteoporosis☆
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Cited by (52)
Altered collagen chemical compositional structure in osteopenic women with past fractures: A case-control Raman spectroscopic study
2021, BoneCitation Excerpt :Such elevations could reflect increased intra-strand stability of the collagen triple helix or increased post-translational hydroxylation of proline residues in collagen [34,35]. While bone and urinary hydroxyproline levels have frequently been used as biomarkers of bone remodeling in postmenopausal women with osteoporosis [95–97], the Hyp/Pro ratio used in this study was not calibrated to provide an absolute measure of proline hydroxylation. Alternatively, the marginal increase in Hyp/Pro ratio could be attributed to indirect effects that are not related to post-translational modifications but to changes in collagen secondary structure.
The nature of osteoporosis
2020, Marcus and Feldman’s OsteoporosisBone Biology and Effects of Pharmaceutical Intervention on Bone Quality
2017, Materials and Devices for Bone DisordersMagnesium intake, bone mineral density, and fractures: Results from the Women's Health Initiative Observational Study
2014, American Journal of Clinical NutritionThe Nature of Osteoporosis
2013, Osteoporosis: Fourth EditionInfluence of chronic alcoholism and oestrogen deficiency on the variation of stoichiometry of hydroxyapatite within alveolar bone crest of rats
2012, Archives of Oral BiologyCitation Excerpt :In cases concerning the changes in the rate of remodelling, with a predominance of the reabsorption process, as occurs in osteoporosis, there is not enough time for osteoblasts to complete the process of mineralization before the bone is reabsorbed prematurely by osteoclasts. These factors could affect the degree of bone mineralization and consequently the Ca/P ratios.6,7,36 In the present study, only the association of alcohol with oestrogenic deficiency was able to significantly decrease the Ca/P ratio in alveolar bone.
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Supported in part by contract N01 AM3-2208 from the National Institutes of Health. A portion of this work was conducted through the Clinical Research Center Facility of the University of Washington, supported by grant #RR-37 from the National Institutes of Health.