Conclusion
From histologic study of 161 cases of acute focal lesions of the gastric mucosa, we would conclude.
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1.
Regardless of etiology, focal gastric lesions are the result of chronic circulatory insufficiency to all the structures of the gastric wall.
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2.
Such circulatory deficiency is part of a generalized insufficiency intensified by intrinsic vascular peculiarities of the stomach, of an anatomical or vasomotor nature.
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3.
Generalized circulatory insufficiency may be established through quantitative, qualitative or vasomotor alterations in the circulatory system.
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References
Reeves, T. B.: Collected Papers of The Mayo Clinic, 10: 3, 1918.
Weech, A. A. and Paige, B. H.:Am. J. Path., 13:249–256, March, 1937.
Nedzel, A. J.:Arch. Path., 26:988–1008, Nov., 1938.
Additional References
Ferguson, A. E.:Am. J. Anat., 42:403–435, 1928.
Robertson, H. E.:J. A. M. A., 112:22, Jan., 1939.
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Read at the Annual 1939 Session of the American Gastro-Enterological Association at Atlantic City.
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Boles, R.S., Riggs, H.E. & Griffiths, J.O. The role of the circulation in the production of peptic ulcer. American Journal of Digestive Diseases 6, 632–636 (1939). https://doi.org/10.1007/BF02996336
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DOI: https://doi.org/10.1007/BF02996336