Abstract
The inflammatory bowel diseases (IBD), ulcerative colitis (UC) and Crohn’s disease (CD) are characterized by recurrent episodes of intestinal inflammation of unknown cause. Although clinical, genetic and pathological differences exist, it is generally agreed that both diseases are a result of an inherited predisposition expressed as an abnormal immune response to exogenous or environmental triggers. The exact nature of the defect in the immune response or the nature of the environmental trigger(s) is not known.
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Streutker, C., Croitoru, K. (2003). Evidence implicating Helicobacter spp. in the pathogenesis of inflammatory bowel disease. In: Hunt, R.H., Tytgat, G.N.J. (eds) Helicobactor pylori. Springer, Dordrecht. https://doi.org/10.1007/978-94-017-1763-2_4
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DOI: https://doi.org/10.1007/978-94-017-1763-2_4
Publisher Name: Springer, Dordrecht
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