Abstract
Creutzfeldt-Jakob disease (CJD), together with its clinical variants, is the more common of the two subacute human spongiform encephalopathies, the other being kuru. The spongiform encephalopathies are slow infections caused by filterable self-replicating agents, the nature of which is not yet fully elucidated. The human diseases resemble three conditions of animals: scrapie of sheep and goats, transmissible mink encephalopathy, and wasting disease of mule deer and elk.
This is a preview of subscription content, log in via an institution to check access.
Access this chapter
Tax calculation will be finalised at checkout
Purchases are for personal use only
Further reading
Gajdusek DC (1977): Unconventional viruses and the origin and disappearance of kuru. Science 197: 943–960
Gajdusek DC (1985): Unconventional viruses causing subacute spongiform encephalopathies. pp 1519–1557. In: “Virology” BN Fields, ed. New York: Raven Press
Merz PA, et al (1984): Infection-specific particle from the unconventional virus diseases. Science 225: 437–440
Oesch B, et al (1985): A cellular gene encodes scrapie PrP27–30 protein. Cell 40: 735–746
Prusiner SB (1982): Novel proteinaceous particles cause scrapie. Science 216: 136–144
Rohwer RG (1984): Scrapie infectious agent is virus-like in size and susceptibility to inactivation. Nature (London) 308: 658–662
Rights and permissions
Copyright information
© 1989 Springer Science+Business Media New York
About this chapter
Cite this chapter
Gibbs, C.J., Asher, D.M. (1989). Creutzfeldt-Jakob Disease. In: Abnormal States of Brain and Mind. Readings from the Encyclopedia of Neuroscience . Birkhäuser, Boston, MA. https://doi.org/10.1007/978-1-4899-6768-8_15
Download citation
DOI: https://doi.org/10.1007/978-1-4899-6768-8_15
Publisher Name: Birkhäuser, Boston, MA
Print ISBN: 978-1-4899-6770-1
Online ISBN: 978-1-4899-6768-8
eBook Packages: Springer Book Archive