Regular Articleβ-Amyloid Fragment 25–35 Causes Mitochondrial Dysfunction in Primary Cortical Neurons
References (39)
- et al.
Interaction of rat brain phosphofructokinase with Alzheimer's beta A4-amyloid
Neurochem. Int.
(1995) - et al.
beta-amyloid fragment 25–35 selectively decreases complex IV activity in isolated mitochondria
FEBS Lett.
(1999) - et al.
Mitochondrial DNA damage as a mechanism of cell loss in Alzheimer's disease
Lab. Invest.
(2000) - et al.
The hepatocellular metabolism of 4-hydroxynonenal by alcohol dehydrogenase, aldehyde dehydrogenase, and glutathione S-transferase
Arch. Biochem. Biophys.
(1995) - et al.
Protein measurement with the Folin Phenol Reagent
J. Biol. Chem.
(1951) - et al.
The inhibitory effects of beta-amyloid on glutamate and glucose uptakes by cultured astrocytes
Brain Res.
(1997) - et al.
Involvement of oxidative stress on the impairment of energy metabolism induced by A beta peptides on PC12 cells: Protection by antioxidants
Neurobiol. Dis.
(1999) - et al.
Cytochrome oxidase from beef heart mitochondria
Methods Enzymol.
(1967) - et al.
Mitochondria and cell death: Mechanistic aspects and methodological issues
Eur. J. Biochem.
(1999) - et al.
Effect of peroxynitrite on the mitochondrial respiratory chain: Differential susceptibility of neurones and astrocytes in primary culture
J. Neurochem.
(1995)
Thiamine-dependent enzyme changes in temporal cortex of patients with Alzheimer's disease
Metab. Brain Dis.
(1990)
Functional mitochondria are required for amyloid beta-mediated neurotoxicity
FASEB J.
(2001)
Beta-amyloid inhibits integrated mitochondrial respiration and key enzyme activities
J. Neurochem.
(2002)
Mitochondrial abnormalities in Alzheimer's disease
J. Neurosci.
(2001)
Regional glucose metabolic abnormalities are not the result of atrophy in Alzheimer's disease
Neurology
(1998)
Impairment of glucose and glutamate transport and induction of mitochondrial oxidative stress and dysfunction in synaptosomes by amyloid beta-peptide: Role of the lipid peroxidation product 4-hydroxynonenal
J. Neurochem.
(1997)
Preparation of succinate cytochrome c reductase, and the cytochrome b-cl particle, and reconstitution of succinate cytochrome c reductase
Methods Enzymol.
(1967)
Brain cytochrome oxidase in Alzheimer's disease
J. Neurochem.
(1992)
Cytotoxic amyloid peptides inhibit cellular 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) reduction by enhancing MTT formazan exocytosis
J. Neurochem.
(1997)
Cited by (0)
- 1
Present address: Lilly Research Centre, Eli Lilly and Company, Erl Wood Manor, Sunninghill Rd., Windlesham, Surrey, GU20 6PH, UK.
- 2
To whom correspondence and reprint requests should be addressed. Fax: +44 20 7833 1016. E-mail: [email protected].
Copyright © 2002 Elsevier Science (USA). All rights reserved.