Regular ArticleGenomic Cloning and Promoter Analysis of the Mouse 105-kDa Heat Shock Protein (HSP105) Gene
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Requirement of Hsp105 in CoCl<inf>2</inf>-induced HIF-1α accumulation and transcriptional activation
2017, Experimental Cell ResearchCitation Excerpt :In CoCl2-treated cells, we observed dose-dependent increases in the expression of Hsp105α (Fig. 1A). However, this was not affected by the knockdown of HIF-1α (Fig. 4C and D), suggesting that CoCl2 may induce the expression of Hsp105α through a HIF-1-independent pathway, such as the HSF1 pathway [37]. In order to examine the interaction between endogenous Hsp105α and CoCl2-induced HIF-1α, cells were treated with 0.25 or 0.5 mM CoCl2 and immunoprecipitation using the anti-human-Hsp105 antibody was performed.
Nmi interacts with Hsp105β and enhances the Hsp105β-mediated Hsp70 expression
2014, Experimental Cell ResearchCitation Excerpt :Hsp70 expression is upregulated by other heat shock proteins, such as Hsp105β, through the Stat signaling pathway [16], which suggests the existence of a chaperone network between these proteins. The Hsp105/110 family members, Hsp105α and Hsp105β are alternatively spliced products derived from an hsp105 gene transcript, and Hsp105β lacks 44 amino acids from Hsp105α [17]. Hsp105α is expressed constitutively and is further induced by various stresses, whereas Hsp105β is specifically expressed during mild heat shock [18–21].
Different localization of Hsp105 family proteins in mammalian cells
2007, Experimental Cell ResearchExamination of the expression of the heat shock protein gene, hsp110, in Xenopus laevis cultured cells and embryos
2006, Comparative Biochemistry and Physiology - A Molecular and Integrative PhysiologyExpression and localization of heat shock factor (Hsf) 1 in the rodent cochlea
2002, Hearing ResearchHSP-CBF is an NF-Y-dependent Coactivator of the Heat Shock Promoters CCAAT Boxes
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To whom correspondence should be addressed at Department of Biochemistry, Kyoto Pharmaceutical University, 5 Nakauchi-cho, Misasagi, Yamashina-ku, Kyoto 607-8414, Japan. Fax: 075-595-4758. E-mail:[email protected].