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CORRECTION article

Front. Neurol., 27 November 2020
Sec. Movement Disorders
Volume 11 - 2020 | https://doi.org/10.3389/fneur.2020.625446
This article is part of the Research Topic Role of Diet, Physical Activity and Immune System in Parkinson’s Disease View all 10 articles

Corrigendum: Gut–Brain Axis: Potential Factors Involved in the Pathogenesis of Parkinson's Disease

This article is a correction to:

  1. Gut–Brain Axis: Potential Factors Involved in the Pathogenesis of Parkinson's Disease

    1. Read original article
\nYin-Xia Chao,, &#x;Yin-Xia Chao1,2,3*†Muhammad Yaaseen Gulam&#x;Muhammad Yaaseen Gulam1†Nicholas Shyh Jenn ChiaNicholas Shyh Jenn Chia1Lei FengLei Feng4Olaf RotzschkeOlaf Rotzschke5Eng-King Tan,,Eng-King Tan1,2,3
  • 1Department of Neurology, National Neuroscience Institute, Singapore, Singapore
  • 2Department of Neurology, Singapore General Hospital, Singapore, Singapore
  • 3Duke NUS Medical School, Singapore, Singapore
  • 4Department of Psychological Medicine, Yong Loo Lin School of Medicine, National University of Singapore, Singapore, Singapore
  • 5Singapore Immunology Network, Agency for Science, Technology and Research, Singapore, Singapore

A Corrigendum on
Gut–Brain Axis: Potential Factors Involved in the Pathogenesis of Parkinson's Disease

by Chao, Y.-X., Gulam, M. Y., Chia, N. S. J., Feng, L., Rotzschke, O., and Tan, E.-K. (2020). Front. Neurol. 11:849. doi: 10.3389/fneur.2020.00849

In the original article, there was an error. The findings cited in Reference Number 43 (Wallen et al., 2020) were inaccurately stated.

A correction has been made to Common factors in the Pathogenesis of PD and Gastrointestinal Disorders, Gut Microbiota, Paragraph 3. The corrected paragraph is shown below:

More recently, Wallen et al. conducted an association study (MWAS) between microbiome and PD using two large datasets. They found that the opportunistic pathogens and carbohydrate-metabolizing probiotics were significantly increased while short-chain fatty acid (SCFA)–producing bacteria were decreased in PD patients (43). These findings will facilitate testing the potential role of some of these pathogens in PD pathogenesis.

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

References

43. Wallen ZD, Appah M, Dean MN, Sesler CL, Factor SA, Molho E, et al. Characterizing dysbiosis of gut microbiome in PD: evidence for overabundance of opportunistic pathogens. NPJ Parkinsons Dis. (2020) 6:11. doi: 10.1038/s41531-020-0112-6

PubMed Abstract | CrossRef Full Text | Google Scholar

Keywords: Parkinson's disease (PD), gut, genetics, microbiome, diet

Citation: Chao Y-X, Gulam MY, Chia NSJ, Feng L, Rotzschke O and Tan E-K (2020) Corrigendum: Gut–Brain Axis: Potential Factors Involved in the Pathogenesis of Parkinson's Disease. Front. Neurol. 11:625446. doi: 10.3389/fneur.2020.625446

Received: 03 November 2020; Accepted: 04 November 2020;
Published: 27 November 2020.

Approved by:

Frontiers Editorial Office, Frontiers Media SA, Switzerland

Copyright © 2020 Chao, Gulam, Chia, Feng, Rotzschke and Tan. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Yin-Xia Chao, chao.yinxia@singhealth.com.sg

†These authors have contributed equally to this work

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.