Immune systems in skin wound healing

Toshikazu Kondo

Article ID: 106
Vol 1, Issue 1, 2017

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References


1. Kondo T, Ishida Y. Molecular pathology of wound healing. Forensic Sci Int 2010; 203(1–3): 93–98. doi: 10.1016/j.forsciint.2010.07.004.

2. Lin ZQ, Kondo T, Ishida Y, et al. Essential invol­vement of IL-6 in the skin wound-healing process as evidenced by delayed wound healing in IL-6-deficient mice. J Leukoc Biol 2003; 73(6): 713–721. doi: 10.1189/jlb.0802397.

3. Ishida Y, Kondo T, Kimura A, et al. Absence of IL-1 receptor antagonist impaired wound healing along with aberrant NF-κB activation and a reci­procal suppression of TGF-β signal pathway. J Immunol 2006; 176(9): 5598–5606. doi: 10.4049/jimmunol.176.9.5598.

4. Mori R, Kondo T, Ohshima T, et al. Accelerated wound healing in tumor necrosis factor receptor p55-deficient mice with reduced leukocyte in­filtration. FASEB J 2002; 16(9): 963–974. doi: 10.1096/fj.01-0776com.

5. Ishida Y, Kondo T, Takayasu T, et al. The essential involvement of cross-talk between IFN-γ and TGF-β in the skin wound-healing process. J Immunol 2004; 172(3): 1848–1855. doi: 10.4049/jimmunol.172.3.1848.




DOI: https://doi.org/10.24294/ti.v1.i2.106

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