Journal of Biological Chemistry
Gene RegulationInhibition of histone H3K27 demethylases selectively modulates inflammatory phenotypes of natural killer cellsEpigenetic modulation of NK cell phenotypes
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This work was supported by Kennedy Trust for Rheumatology Research, Arthritis Research UK Program Grant 20522, the National Institute of Health Research Oxford Biomedical Research Unit, Cancer Research UK, and the Rosetrees Trust. This work is supported in part by Medical Research Council CGAT Program Grant G1000902 (to A. C.). The Structural Genomics Consortium is a registered charity (Number 1097737) that receives funds from Abbvie, Bayer Healthcare, Boehringer Ingelheim, the Canadian Institutes for Health Research, the Canadian Foundation for Innovation, Eli Lilly and Company, Genome Canada, the Ontario Ministry of Economic Development and Innovation, Janssen, the Novartis Research Foundation, Pfizer, Takeda, and the Wellcome Trust. The research leading to these results has received funding from the People Programme (Marie Curie Actions) of European Union Seventh Framework Programme FP7/2007–2013 under Research Executive Agency (REA) Grant Agreement 609305. The authors declare that they have no conflicts of interest with the contents of this article.
This article contains supporting information, Tables S1–S3, and Figs. S1–S3.
RNA-seq, ATACs-eq, and ChIP-seq data sets were deposited with the GEO database under accession number GSE89669.