References
Arikkath J and Campbell KP (2003) Auxiliary subunits: essential components of the voltage-gated calcium channel complex. Curr Opin Neurobiol 13: 298–307.
Flucher BE, Andrews SB and Daniels MP (1994) Molecular organization of transverse tubule/sarcoplasmic reticulum junctions during development of excitation-contraction coupling in skeletal muscle. Mol Biol Cell 5: 1105–1118.
Freise D, Held B, Wissenbach U, Pfeifer A, Trost C, Himmerkus N, Schweig U, Freichel M, Biel M, Hofmann F, Hoth M and Flockerzi V (2000) Absence of the gamma subunit of the skeletal muscle dihydropyridine receptor increases L-type Ca2+ currents and alters channel inactivation properties. J Biol Chem 275: 14476–14481.
Gregg RG, Messing A, Strube C, Beurg M, Moss R, Behan M, Sukhareva M, Haynes S, Powell JA, Coronado R and Powers PA (1996) Absence of the beta subunit (cchb1) of the skeletal muscle dihydropyridine receptor alters expression of the alpha 1 subunit and eliminates excitation-contraction coupling. Proc Natl Acad Sci USA 93: 13961–13966.
Takeshima H, Iino M, Takekura H, Nishi M, Kuno J, Minowa O, Takano H and Noda T (1994) Excitation-contraction uncoupling and muscular degeneration in mice lacking functional skeletal muscle ryanodine-receptor gene. Nature 369: 556–559.
Tanabe T, Beam KG, Powell JA and Numa S (1988) Restoration of excitation-contraction coupling and slow calcium current in dysgenic muscle by dihydropyridine receptor complementary DNA. Nature 336: 134–139.
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Obermair, G.J., Kugler, G. & Flucher, B.E. The Role of the Calcium Channel α2δ-1 Subunit in Skeletal Muscle. J Muscle Res Cell Motil 25, 239–240 (2004). https://doi.org/10.1023/B:JURE.0000038361.47060.fe
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DOI: https://doi.org/10.1023/B:JURE.0000038361.47060.fe