Short communicationBilateral dislocation of the knee with rupture of both patellar tendons. A case report
Introduction
Bilateral traumatic rupture of the patellar tendon is an uncommon injury [1]. Patients with chronic systemic diseases such as rheumatoid arthritis, systemic lupus erythematosus, chronic renal disease, and gout were involved in the majority of reported cases of bilateral patellar tendon ruptures [2]. Corticosteroid medication has also been implicated. Although a few cases of bilateral traumatic rupture have been described in the literature, to the best of author's knowledge no case associated with bilateral knee dislocation have been reported previously. The following might be the first such case.
Section snippets
Case report
A 30-year-old male motorcyclist had a road traffic accident. He was riding a motor bike in the countryside, hit by a car. There were no accompanying injuries, other than bilateral complete dislocation of the knee and a fracture of fibula on the left side. His feet were cool and no popliteal or ankle pulses were detected. His blood pressure and pulse were within normal limits. No other injuries and skin lesions except bruising were noted on physical examination. Direct radiographs revealed
Discussion
Although rarely reported in the literature, patellar tendon rupture is frequently seen in the sporting population, and its usual straight forward treatment makes the injury quite satisfying to manage [3]. Most patellar tendon ruptures occur at the inferior pole as an avulsion or, less commonly, as a mid-substance rupture. Many factors may cause weakness of the tendon or muscle rendering them susceptible to spontaneous rupture. Aging causes loss of tissue elasticity, decreased tissue perfusion
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2016, International Emergency NursingCitation Excerpt :Most injuries are incurred as a result of marked hyper-flexion of the knee during sporting activity (Bhargava et al., 2004; Moretti et al., 2014), with the cumulative effect of microfractures, usually at the proximal insertion of the tendon, being thought to contribute to this increase in risk in otherwise healthy individuals (Bhargava et al., 2004; Greis et al., 2005; Kumar et al., 2010; Lu et al., 2012). In addition patients who have been administered steroidal injections to the joint, particularly on a repeated basis, may be at risk of rupture (Ozkan et al., 2006; Sibley et al., 2012). Other reports of rupture demonstrate an increased prevalence in patients with systemic illnesses pre-disposing to tendinopathy or for which they are receiving steroid therapy.
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