Original ReportInhibition of Carbonic Anhydrase Augments GABAA Receptor-Mediated Analgesia via a Spinal Mechanism of Action
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Key words
Gamma-aminobutyric acid
neuron-specific K+-Cl– cotransporter
neuropathic pain
carbonic anhydrase
spinal analgesia
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This work was supported by funds from the Rita Allen Foundation (T.J.P.), National Institutes of Health grant NS065926 (T.J.P.), The Academy of Finland (K.K.), The Sigrid Jusélius Foundation (K.K.), The Scan | Design Foundation By Inger & Jenns Bruun (T.J.P. and K.K.) and the DFG grant HU 800/5-1 (C.A.H.). T.J.P. is a Rita Allen Foundation Scholar in Pain.
The authors declare no conflicts of interest.
Copyright © 2014 American Pain Society. Published by Elsevier Inc. All rights reserved.