Reflection and ReactionGood clinical practice in neurorehabilitation
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Cited by (9)
Gait disorders
2013, Handbook of Clinical NeurologyCitation Excerpt :The close similarity of gait between parkinsonian patients and children who have not yet developed a plantigrade gait has led to the suggestion that an immature pattern may reappear in Parkinson disease as a result of deficits in the neuronal circuits controlling plantigrade locomotion (Forssberg et al., 1984). Furthermore, coordination between the lower limbs as well as between upper and lower limbs is impaired during walking in parkinsonian compared to age-matched healthy subjects (Dietz and Colombo, 1996; Winogrodzka et al., 2005; Dietz, 2006; Dietz and Michel, 2008). Defective coordination of upper and lower limbs (Swinnen et al., 1997; Winogrodzka et al., 2005), in combination with reduced arm swing (Carpinella et al., 2007), and abnormal postural reactions to voluntary movements (Rogers et al., 1987) might contribute to the impaired performance of obstacle avoidance locomotion in Parkinson disease (van Hedel et al., 2006).
Sensor-driven four-channel stimulation of paretic leg: Functional electrical walking therapy
2009, Journal of Neuroscience MethodsReply from the author [2]
2006, NeurologyClinical aspects for the application of robotics in locomotor neurorehabilitation
2016, Neurorehabilitation Technology, Second EditionTraining with robot-applied resistance in people with motor-incomplete spinal cord injury: Pilot study
2015, Journal of Rehabilitation Research and DevelopmentFeedback and feedforward locomotor adaptations to ankle-foot load in people with incomplete spinal cord injury
2010, Journal of Neurophysiology