ω-Substituted alkyl carboxylic acids as antidiabetic and lipid-lowering agents
References (36)
- et al.
J. Diab. Comp.
(1995) - et al.
Metabolism
(1991) - et al.
J. Biol. Chem.
(1996) - et al.
J. Biol. Chem.
(1995) Curr. Ther. Res.
(1997)- et al.
J. Lipid Res.
(1983) - et al.
J. Lipid Res.
(1979) - et al.
Metabolism
(1977) Diabetologia
(1992)- et al.
Diabetes/Metab. Rev.
(1997)
Diabetes Care
(1991)
Diabetes
(1988)
Diabetes
(1996)
Diabetes Stoffwechsel
(1992)
Diabetes Care
(1990)
Diabetes
(1983)
Arzneim. Forsch.
(1990)
Diabetes
(1990)
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