Identification and mapping of a mouse gene influencing cerebellar folial pattern
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Cited by (28)
Spontaneous malformations of the cerebellar vermis: Prevalence, inheritance, and relationship to lobule/fissure organization in the C57BL/6 lineage
2015, NeuroscienceCitation Excerpt :As shown in Fig. 1C, D, the organization of the cerebellar vermis varies substantially, with some strains having as many as 13 lobules and 11 fissures (Fig. 1A, B). For example, approximately 10% of C57BL/6J mice display the presence of an additional cerebellar fissure known as the ICF Inouye and Oda, 1980) and a genetic locus responsible for this trait has been mapped to chromosome 4 (Neumann et al., 1990; Garretson and Neumann, 1993). In contrast, 100% of DBA/2J inbred mice were previously shown to exhibit an ICF (Neumann et al., 1990).
Segmentation of the C57BL/6J mouse cerebellum in magnetic resonance images
2012, NeuroImageCitation Excerpt :This review presents the gross anatomy, lobular organization, and histology of the cerebellum, and the authors summarize the distinctive features of the C57BL/6J cerebellum compared to that of other mouse strains. For example, when compared with the DBA/2T mouse, the C57BL/6J lacks an interculminate fissure, which results in incomplete separation of lobules 4 and 5, and, in addition, the boundary between lobules 1 and 2 is difficult to define (Neumann et al., 1990; Neumann et al., 1993). The proposed protocol for the segmentation of the cerebellum aims to support a consistent approach to quantitative studies of regional volumes in the cerebellum.
Cerebellum
2012, The Mouse Nervous SystemCerebellum
2011, The Mouse Nervous SystemHow Many Ways Can Mouse Behavioral Experiments Go Wrong? Confounding Variables in Mouse Models of Neurodegenerative Diseases and How to Control Them
2010, Advances in the Study of BehaviorCitation Excerpt :In this way, the genetic regulation of the neural bases of behavior can be examined (Havekes and Abel, 2009; Luo et al., 2008). A number of effects of genetic manipulation have been demonstrated in the brains of mice, including changes in the size and weight of the neocortex (Eleftheriou et al., 1975; Shimada, 1999), the cerebellum (Neumann et al., 1990; Wahlsten et al., 2003a), the hippocampus (Peirce et al., 2003; Thompson et al., 2008; Wahlsten et al., 2003a), the corpus callosum (Kusek et al., 2007; Wahlsten, 1982a; Wahlsten et al., 2003a), and the medial preoptic-anterior hypothalamus (A.E. Brown et al., 1999; Mathieson et al., 2000). There are also genetic influences on neurotransmitter activity (Ebel et al., 1987), the receptors for neurotransmitters (Ng et al., 1994), neurosteroid levels (Tagawa et al., 2006), neuropeptide distribution (Mathieson et al., 2000; Weitemier et al., 2005), and neuroendocrine function (Lee et al., 2007).
Abnormality in the cerebellar folial pattern of C57BL/6J mice
2005, Neuroscience Letters