Research reportResponses of the suprachiasmatic nucleus to retinohypothalamic tract volleys in a slice preparation of the mouse hypothalamus
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Imaging the Role of Myelin in Concussion
2018, Neuroimaging Clinics of North AmericaCitation Excerpt :Although myelin decompaction may not be as severe an injury as myelin degeneration, there is still reason to view these changes as a serious mTBI disorder, because they lead to reductions in action potential conduction.59 Myelinated axons in mice and rats have been found to conduct at a rate of 2.4 m/s, which is significantly greater than the unmyelinated rate of 0.4 m/s.60,61 Axons of mice with decompacted myelin conduct at a rate of about 1.05 m/s.59 This difference represents a reduction of more than half the conduction rate, and could in turn be responsible for some of the known cognitive deficits following mTBI,62 such as affected memory, attention, processing speed, and executive functioning. As discussed earlier, myelin decompaction in mTBI is likely to be caused by secondary mechanisms, such as oxidative stress.25
In vitro stimulation of c-Fos protein expression in the suprachiasmatic nucleus of hypothalamic slices
1996, Molecular Brain ResearchNeurochemical organization of circadian rhythm in the suprachiasmatic nucleus
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Present address: M. Menaker, Department of Biology, University of Virginia, Charlottesville, VA 22901, U.S.A.