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Functional preservation of hippocampal CA1 by low-dose GYKI-52466 preconditioning in a rat model of hypoxic-ischemic brain injury
2015, Brain ResearchCitation Excerpt :In the present study, LTP was completely abolished in the few ipsilateral hippocampal slices that could be recovered from HI animals, and markedly reduced in contralateral slices up to 90 days post-HI, without any apparent change in LTD induction. Again, the most likely explanation for this contralateral LTP deficit probably relates to the loss of commissural fibers from the ipsilateral (stroke) side of the brain (Wheal et al., 1983). As such, marked reductions in net synaptic density would reduce cooperative/associative interactions necessary for strong LTP induction (Bliss and Collingridge, 1993; McNaughton, 2003).
Inhibition of hippocampal LTP by ginkgolide B is mediated by its blocking action on PAF rather than glycine receptors
2004, Neurochemistry InternationalAn efficient method for studying short-term plasticity with random impulse train stimuli
2002, Journal of Neuroscience MethodsFunction of synapses in the CA1 region of the hippocampus: Their contribution to the generation or control of epileptiform activity
1989, Comparative Biochemistry and Physiology -- Part A: Physiology
Copyright © 1983 Published by Elsevier B.V.