Micronutrient status in patients receiving home parenteral nutrition
References (11)
- et al.
Manganese toxicity in children receiving long-term parenteral nutrition
Lancet
(1996) - et al.
Efficacy of water-soluble vitamin E in the treatment of vitamin E malabsorption in short-bowel syndrome
Am J Clin Nutr
(1994) - et al.
Prognosis of patients with non-malignant chronic intestinal failure receiving long-term home parenteral nutrition
Gastroenterology
(1995) Recommended Dietary Allowances
(1989)- et al.
Blood levels of long-term adult home total parenteral nutrition patients: the efficacy of the AMA-FDA parenteral multivitamin preparation
JPEN
(1985)
Cited by (64)
ESPEN practical short micronutrient guideline
2024, Clinical NutritionESPEN micronutrient guideline
2022, Clinical NutritionCitation Excerpt :It also depends on the availability of the gastrointestinal tract, and on the demonstration of selenium-containing biological fluids losses (e.g. in burns or during continuous renal replacement therapy). The basic requirement to normalize plasma selenium during home PN in patients without inflammation is about 60–100 μg/d [284–290]. Certain groups may have higher requirements:
Methods for assessment of biotin (vitamin B<inf>7</inf>)
2018, Laboratory Assessment of Vitamin StatusIntestinal Absorption of Water-Soluble Vitamins: Cellular and Molecular Mechanisms
2018, Physiology of the Gastrointestinal Tract, Sixth EditionThe Sodium/Multivitamin Transporter. A Multipotent System with Therapeutic Implications.
2015, Vitamins and HormonesCitation Excerpt :In particular, biotin deficiency during pregnancy has been shown to cause growth retardation of the embryo, congenital malformation, and even death (Watanabe, 1983, 1996). Deficiency of biotin has been observed in patients with inborn errors of biotin metabolism (Sweetman & Nyhan, 1986; Wolf, 2001) and in patients on long-term therapy with anticonvulsant drugs (Krause et al., 1988), or in those on long-term parenteral nutrition (Forbes & Forbes, 1997), as well as in patients with seboric dermatitis and Leiner's disease (Nisenson, 1957). It has been shown that biotin therapy rapidly improves skin and other congenital abnormalities (Miller, 1989; Mock, 1991; Tsao & Kien, 2002).