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Mechanism of cerebellar function studied using mutant mice

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References

  1. Kashiwabuchi N, Ikeda K, Araki K, Hirano T, Shibuki K, Takayama C, et al. Disturbed motor coordination, Purkinje cell synapse formation and cerebellar long-term depression of mice defective in the d2 subunit of the glutamate receptor channel. Cell. 1995;81:245–52.

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  2. Yoshida T, Katoh A, Ohtsuki G, Mishina M, Hirano T. Oscillating Purkinje neuron activity causing involuntary eye movement in a mutant mouse deficient in the glutamate receptor δ2 subunit. J Neurosci. 2004;24:2440–8.

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  3. Katoh A, Yoshida T, Himeshima Y, Mishina M, Hirano T. Defective control and adaptation of reflex eye movements in mutant mice deficient in either the glutamate receptor δ2 subunit or Purkinje cells. Eur J Neurosci. 2005;21:1315–26.

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Authors and Affiliations

  1. Department of Biophysics, Graduate School of Science, Kyoto University, Kyoto, Japan

    Tomoo Hirano & Yoshida Takashi

  2. CREST, JST, Kawaguchi, Saiatama, Japan

    Tomoo Hirano, Yoshida Takashi & Kazuo Funabiki

  3. HMRO, Graduate School of Medicine, Kyoto University, Kyoto, Japan

    Kazuo Funabiki

Authors
  1. Tomoo Hirano
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  2. Yoshida Takashi
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  3. Kazuo Funabiki
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Hirano, T., Takashi, Y. & Funabiki, K. Mechanism of cerebellar function studied using mutant mice. Cerebellum 5, 299–300 (2006). https://doi.org/10.1007/BF02835432

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  • DOI: https://doi.org/10.1007/BF02835432

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