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Kinesin Activation Drives the Retrieval of Secretory Membranes Following Secretion in Rabbit Lacrimal Acinar Cells

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Lacrimal Gland, Tear Film, and Dry Eye Syndromes 2

Part of the book series: Advances in Experimental Medicine and Biology ((AEMB,volume 438))

Abstract

Recent studies in the lacrimal gland have focused on how changes in membrane trafficking may contribute to the development of tear insufficiency and the development of autoimmunity in the lacrimal gland. We have focused on resolving the roles that microtubule-based vesicle transport and the individual motor proteins, kinesin and cytoplasmic dynein, play in facilitating membrane-trafficking events in lacrimal acini.

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References

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Hamm-Alvarez, S.F., da Costa, S.R., Sonee, M., Warren, D.W., Mircheff, A.K. (1998). Kinesin Activation Drives the Retrieval of Secretory Membranes Following Secretion in Rabbit Lacrimal Acinar Cells. In: Sullivan, D.A., Dartt, D.A., Meneray, M.A. (eds) Lacrimal Gland, Tear Film, and Dry Eye Syndromes 2. Advances in Experimental Medicine and Biology, vol 438. Springer, Boston, MA. https://doi.org/10.1007/978-1-4615-5359-5_25

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  • DOI: https://doi.org/10.1007/978-1-4615-5359-5_25

  • Publisher Name: Springer, Boston, MA

  • Print ISBN: 978-1-4613-7445-9

  • Online ISBN: 978-1-4615-5359-5

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