Abstract
Transient global amnesia (TGA) is a common paroxysmal disorder of episodic memory. The aetiology of TGA is still unknown. Brain ischaemia, migraine, epileptic seizure, venous congestion and psychological disturbances have been proposed as pathological mechanisms. Moreover, different precipitating events are recognised in most TGA patients including physical activity, severe emotional stress, painful experiences, immersion in cold water and sexual intercourse. We describe a 54-year-old woman who presented a TGA immediately after right-left shunt of saline contrast during the execution of transoesophageal echocardiography. Aetiopathological considerations for this uncommon presentation are discussed.
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References
Lewis SL (1998) Aetiology of transient global amnesia. Lancet 352:397–399
Fisher CM (1982) Transient global amnesia. Precipitating activities and other observations. Arch Neurol 39:605–608
Richardson RS, Leek BT, Wagner PD, Kritchevsky M (1998) Transient global amnesia: a complication of incremental exercise testing. Med Sci Sports Exerc 30[10 Suppl]:S403–S405
Klotzsch C, Sliwka U, Berlit P, Noth J (1996) An increased frequency of patent foramen ovale in patients with transient global amnesia. Analysis of 53 consecutive patients. Arch Neurol 53:504–508
Sander D, Winbeck K, Etgen T et al (2000) Disturbance of venous flow patterns in patients with transient global amnesia. Lancet 356:1982–1984
Akkawi NM, Agosti C, Rozzini L et al (2001) Transient global amnesia and disturbance of venous flow patterns. Lancet 357:957
Guillery B, Desgranges B, de la Sayette V et al (2002) Transient global amnesia: concomitant episodic memory and positron emission tomography assessment in two additional patients. Neurosci Lett 325:62–66
Strupp M, Bruning R, Wu RH et al (1998) Diffusion-weighted MRI in transient global amnesia: elevated signal intensity in the left mesial temporal lobe in 7 of 10 patients. Ann Neurol 43:164–170
Sedlaczek O, Hirsch JG, Grips E et al (2004) Detection of delayed focal MR changes in the lateral hippocampus in transient global amnesia. Neurology 62:2165–2167
Winbeck K, Etgen T, von Einsiedel HG et al (2005) DWI in transient global amnesia and TIA: proposal for an ischaemic origin of TGA. J Neurol Neurosurg Psychiatry 76:438–441
Nakada T, Kwee IL, Fujii Y, Knight RT (2005) High-field, T2 reversed MRI of the hippocampus in transient global amnesia. Neurology 64:1170–1174
Cianfoni A, Tartaglione T, Gaudino S et al (2005) Hippocampal magnetic resonance imaging abnormalities in transient global amnesia. Arch Neurol 62:1468–1469
Menéndez González M, Rivera MM (2006) Transient global amnesia: increasing evidence of a venous etiology. Arch Neurol 63:1334–1336
Quinette P, Guillery-Girard B, Dayan J et al (2006) What does transient global amnesia really mean? Review of the literature and thorough study of 142 cases. Brain 129:1640–1658
Meissner I, Whisnant JP, Khandheria BK et al (1999) Prevalence of potential risk factors for stroke assessed by transesophageal echocardiography and carotid ultrasonography: the SPARC study. Stroke prevention: assessment of risk in a community. Mayo Clin Proc 74:862–869
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Profice, P., Rizzello, V., Pennestrì, F. et al. Transient global amnesia during transoesophageal echocardiogram. Neurol Sci 29, 477–479 (2008). https://doi.org/10.1007/s10072-008-1034-y
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DOI: https://doi.org/10.1007/s10072-008-1034-y